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RAGE does not contribute to renal injury and damage upon ischemia/reperfusion-induced injury

Dessing, Mark C. ; Pulskens, Wilco P. ; Teske, Gwendoline J. ; Butter, Loes M. ; Poll, Tom van der ; Yang, Huan ; Tracey, Kevin J. ; Nawroth, Peter P. ; Bierhaus, Angelika ; Florquin, Sandrine ; Leemans, Jaklien C.

In: Journal of innate immunity, 4 (2012), Nr. 1. pp. 80-85. ISSN 1662-811X

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Abstract

The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury. Copyright (C) 2011 S. Karger AG, Basel

Document type: Article
Journal or Publication Title: Journal of innate immunity
Volume: 4
Number: 1
Publisher: S. Karger AG
Date Deposited: 12 Jan 2015 13:55
Date: 2012
ISSN: 1662-811X
Page Range: pp. 80-85
Faculties / Institutes: Medizinische Fakultät Heidelberg > Medizinische Universitäts-Klinik und Poliklinik
DDC-classification: 610 Medical sciences Medicine
Controlled Keywords: Ischämie, Niere
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